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Medicine: Diagnosing MS: Understanding MS: A
Case of Definition by Dr. Franz Schelling Neurologist Dornbirn, Austria Wish to understand MS? .... Truly? - which one? Faceless MS?
Clinically-defined CDMS? Nerve dysfunctions, deemed diagnostic, provided they: • Have no known
cause, • Persist, in between
pause, for set periods of time. Devoid of substance, of form, of any physical or organic
property, this ward and consulting room diagnosis of multiple sclerosis is
ultimately nothing but red flags of what it must not be mistaken for, a
widely adopted smokescreen for diverse changes of an untraced nature. Inflammatory
demyelinating or 'ADEM-EAE' MS? Natural acute disseminated encephalomyelitis
(ADEM) and its experimental allergic counterpart EA[D]E[M] is
millet to pea-sized longish foci, or rather sleeves, formed around blood
vessels in the course of inflammatory reactions. These mark the spread of
some circulated agent(s). A report on such a condition (Rindfleisch 1863) led to confusion
in discussing this micro-metastatic form of pathology in a context of not
specified observations of an active injurious process of spinal and cerebral
sclerosis attributed to Rokitansky. A MS-specific inflammatory demyelination, agent, or MS case set
apart by what marks ADEM and EAE has never been identified. To the contrary, a circulated agent is unable to invade the spinal
cord and the brain in the ways MS lesions spread, as first noted by
Steiner in 1931. Another red herring? At any rate there is nobody who ever has
shown this CDMS associated 'ADEM-EAE' [MS] to be more than a clinically
convenient explanatory construct. Distinct MS nature in the
brain and spinal cord? From Carswell (1831) to Oppenheim (1978) autopsies
disclosed more and more findings proper to spinal MS. In cerebral MS radically different MS specific traits were
pictured at post mortem first by Charcot (1866) and since 1981 routinely,
serially on MRI. Puzzling in themselves, these findings resist being fitted
together into a coherent whole unless they are studied separately. Thereby emerges a
distinct picture as to how cerebral and spinal MS evolve. Fully appreciating the two natures of MS will require: • An acquaintance with
some clearly shown and yet widely missed facts: https://medium.com/@franzschelling/open-letter-to-cedric-s-raine-bc1df79b106d, • Riddance of
misleading doctrines: https://medium.com/@franzschelling • Facing up to the
ways in which brain and cord lesions actually advance: www.ms-info.net Ridiculous? But how else can we comprehend the fact that: (A) Venous wetterwinkel/corpus callosum MS lesions mirror those
of traumatic brain injury (TBI): https://dl.dropboxusercontent.com/u/66292082/MS%20WETTERWINKEL%20LESIONS%20PARALLEL%20TRAUMATIC%20BRAIN%20INJURY.pdf (B) Mooring lesions of traumatic spinal cord injury (TSCI) so
closely mimic spinal MS? (C) These changes can only be conceived as a concussive
injuring of the brain and/or spinal core by sporadic overly intensive
expansions of veins in either the brain or spinal canal? Any MRI time-lapse series of MS patients that shows: · A 'cockscomb' (Heckl 1994) lesion growth along
and off the outer angle of a lateral ventricle, · Splashy (Steiner 1931) single-hit plaques (Raine
1997), hemorrhages spread in the same way · Engorgement, then shrinking of local veins… · A sclerosing process invading the spinal cord
from insertion zones of mainly the denticulate ligament…
Specific form of CCSVI
primed MS? The kind of brain and cord damages that relates to stenoses of
internal jugular or prevertebral veins is, quite paradoxically, nowhere
discussed. The stenoses of these veins further three principally different
kinds of injury: · A swelling, at times dotted with
ring-bleedings, that involves the obstructed vein's entire periphery; usually
the result of complete obstructions of intracranial segments of venous pathways
of the brain. · Farther off the skull, hindrances to the
cerebral venous flow predispose to a different kind of events, rather known to
occur in the legs: Compression of an engorged suprastenotic vein length drives
blood back in direction of, and eventually into, especially exposed veins of
the brain. The routing of such a flow at the confluence of sinuses attains
critical significance. · The retrograde emptying of blood dammed up in
pre- or perivertebral veins hardly ever overburdens spinal cord veins
themselves; abrupt engorgements of some part of the wide, dense mesh of
epidural veins instead seek to dislocate the dural sac's subjacent
contents. In obstructions of internal jugular veins it has to be found out
whether there is: (i) A diffuse stasis that encompasses the obstructed vessel's
entire periphery, or (ii) A picture of recurrent impacts exerted on the part of first
its larger and later smaller collecting veins. In obstructions of major
prevertebral veins (inferior cava, azygos, left renal, or iliac vein) or big
deep cervical veins, (iii) A progressive damaging of the cord via insertions of its
mooring in the spinal dural sac has to be taken into account. Not
granting attention to these circumstances, the interventions for CCSVI cannot
be expected to surpass their experimental stage. In summation: Treating MS patients not knowing what they are
affected by denies them reliable benefits and entails an unwarrantable risk of
aggravating their misery. About the
author: The town of Linz in Austria was being hit by an air
raid when Franz Alfons Schelling was born there on March 31, 1945. Early on,
the boy was gripped by a passion for pursuing problematic issues to their concrete sources. Since 1970, the
year during which he earned the status of medical doctor, Dr. Schelling has confronted
questions concerning the genesis of infantile hydrocephaly. Soon after that, he took a special interest in
clarifying the significance of the differences in the development of the
emissary veins, the venous outlets of the human skull. He started this work in 1973 at the Institute of Anatomy of the University
of Innsbruck, and pursued it during his final two years of internship in Linz. Finally,
in 1978, during the third year of his surgery practice, he arrived at a preliminary conclusion in presenting
his comprehensive evaluation of the anatomy of emissary veins. Returning in 1981 to neurosurgical training at the
Regional Hospital for Nervous Diseases of Salzburg, Dr. Schelling wanted to
clarify to what extent the cranial venous outlets’ global conductivity has an
influence upon the individual cerebrospinal fluid pressure. Thus, he found that
unusually large cranial venous outlets were to be observed mainly in the
radiographies of patients who were thought to be suffering from multiple
sclerosis. As the findings seemed to be caused by a continual repetition of
massive venous flow reversal, they aroused the suspicion that corresponding
back-jets could also be involved in the causation of multiple sclerosis. Unfortunately,
his subsequent working periods in the Neurological and Radiological Departments
of the Regional Hospital of Voralberg brought no substantial advance beyond
that state of affairs. There was no access to post mortem examinations which
would have allowed Dr. Schelling to discover the preconditions for the particular
back-jets’ recurrence, nor was there any chance to produce a flow-mapping which
might have traced the back-jets themselves. In 1985 he therefore returned to
his surgery practice, resigning himself, for the next dozen years, to a study
of the literature on multiple sclerosis. To his astonishment, Dr. Schelling discovered that
the characteristics which proved peculiar to specific instances of multiple
sclerosis could thoroughly, easily and only be explained by considering the
effects which recurring venous back-jets into definite venous drainage of the
brain’s respective spinal canal were bound to produce. He thus became convinced
that multiple sclerosis was to be overcome not by ever more drug trials, but in
soberly considering to which interplay of forces and structures the multiple
sclerosis-specific lesion developments were due. Harassed, since 1997, by a state of
nervous exhaustion which put an end to his medical practice, Dr. Schelling yet
enhanced his effort at pointing a way out of the blind alley into which
multiple sclerosis research has maneuvered itself. Helped by his two sons
Bernhard and Matthias, he was eventually granted, in 2002, relief in the
presentation of his e-book Multiple Sclerosis: the Image and 1st Message at www.multiple-sclerosis-abc.org. [ BWW Society Home Page ] © 2015 The Bibliotheque: World Wide Society |